Cardiogen

Discover Cardiogen, Explore its benefits on cardiomyocyte regeneration, anti-fibrosis, and advanced cardiovascular research. Purity: 99%

Product Description

     Cardiogen is a synthetic short-chain peptide bioregulator specifically engineered to target the cardiovascular system and support myocardial tissue repair. Developed as part of the pioneering research into “Khavinson Peptides,” Cardiogen is a tetrapeptide consisting of four amino acids with the sequence Alanine-Glutamic Acid-Aspartic Acid-Arginine (Ala-Glu-Asp-Arg, or AEDR). It is highly valued in cardiovascular biology and anti-aging research for its precise, tissue-specific affinity for heart cells.

Product benefits

Targeted Cardiomyocyte Regeneration: Cardiogen exhibits a powerful, tissue-specific affinity for the cardiovascular system. In pre-clinical testing, it directly stimulates the proliferation of heart muscle cells (cardiomyocytes), actively promoting tissue renewal and repair in both young and aging cardiac models. 

Inhibition of Cardiac Fibrosis & Scarring: It plays a critical role in preventing unfavorable cardiac remodeling following injury. By regulating fibroblast activity, Cardiogen helps inhibit the excessive development of scar tissue in the myocardium, preserving heart wall elasticity and long-term functional integrity.

Protection Against Cellular Apoptosis: Cardiogen helps shield heart cells from severe physiological stress, ischemia, and hypoxia. It suppresses programmed cell death (apoptosis) in healthy tissue, an effect heavily correlated with its ability to down-regulate the p53 cellular stress pathway.

Epigenetic Genomic Protection: Operating as a precise short-chain bioregulator, Cardiogen interacts directly with DNA to protect cellular strands from enzymatic degradation and endonuclease-driven hydrolysis. This action restores youthful protein synthesis and maintains optimal genomic stability inside cardiovascular tissues.

Dual-Action Onco-Modulatory Potential: Beyond cardiac care, research shows Cardiogen exhibits a highly unique, context-dependent mechanism in oncological models. In transformed cell lines, it has been shown to induce localized tumor-cell apoptosis and hemorrhagic necrosis via the tumor’s specific vascular network, making it a valuable asset for multi-disciplinary laboratory studies.